Probiotics for Body Odor: What the Research Actually Shows
From the gut-skin axis to trimethylaminuria—an evidence-based look at how probiotics address odor at its source
Body odor is one of those topics people quietly research but rarely talk about. It's also one where conventional advice—shower more, switch deodorants, avoid garlic—only scratches the surface of what's actually happening biologically. Because the truth is, body odor is not primarily a hygiene problem. It's a microbiome problem.
Both your skin and your gut harbor microbial ecosystems that directly shape the compounds your body releases into sweat and breath. When those ecosystems are out of balance, the volatile molecules they generate can be unusually pungent. This is why two people with identical hygiene routines can smell completely different—and why some people struggle with persistent odor despite doing everything "right."
Probiotics enter this picture through a growing body of research on the gut-skin axis and the axillary microbiome. Certain strains found in MicroBiome Restore have been studied specifically for their ability to shift microbial populations in ways that reduce odor-causing compounds. This article examines what the peer-reviewed science says—honestly, without overpromising—and explains how the strains in our formula connect to these mechanisms.
We also cover trimethylaminuria (TMAU), a metabolic condition involving pathological body odor with a documented gut microbiome component, where emerging research points to probiotic-adjacent strategies as a meaningful part of management.
Key Takeaways
- Sweat itself is odorless. Body odor is produced when skin-resident bacteria metabolize sweat compounds into volatile organic compounds (VOCs) such as thioalcohols, volatile fatty acids, and ammonia.[1]
- The gut-skin axis is real. What happens in your gut influences systemic inflammation, barrier function, and the types of metabolites circulating in your bloodstream—all of which can affect what is excreted through sweat.[2]
- Lactobacillus delbrueckii subsp. bulgaricus (L. bulgaricus) significantly reduced axillary odor and decreased Corynebacterium abundance in a clinical study of patients with axillary osmidrosis.[3]
- A topical Lactobacillus cream decreased malodor-producing bacteria in human armpits and downregulated genes encoding odor-producing enzymes, providing proof-of-concept that Lactobacillus species can rebalance the axillary microbiome.[4]
- Lactobacillus rhamnosus is the most efficient probiotic strain identified for reducing plasma TMAO levels in both human and animal studies, according to a 2022 systematic review.[5]
- Lactobacillus paracasei-derived postbiotics significantly reduced TMA—the compound responsible for fish odor syndrome—in both in vitro and in vivo models.[6]
- Oral probiotics can positively influence the skin microbiome via the gut-skin axis, reducing inflammatory markers and supporting barrier integrity in ways relevant to odor control.[7]
Why Body Odor Happens: A Microbiome Explanation
The conventional framing—that body odor is caused by sweat—is only half the story. Sweat itself, whether from eccrine glands (distributed across the whole body) or apocrine glands (concentrated in the armpits, groin, and around nipples), is essentially odorless when it leaves the body.[1] The characteristic odors we associate with perspiration emerge downstream, as a result of bacterial metabolism.
Your skin hosts a dense microbial community. In the axillary region alone, bacteria metabolize apocrine sweat secretions into an array of volatile compounds. The key odor-producing bacteria and their chemical byproducts have been identified with increasing precision over the past decade:
The Microbial Chemistry of Armpit Odor
Corynebacterium species are the primary drivers of the characteristic pungent, cheese-like odor associated with body odor. They produce volatile fatty acids—especially 3-methyl-2-hexenoic acid (3M2H)—through enzymatic cleavage of odorless glutamine-conjugated precursors secreted by apocrine glands.[1] The greater the relative abundance of Corynebacterium in your axillary microbiome, the stronger the resulting odor.
Staphylococcus hominis produces pungent sulfur-containing thioalcohols, including 3-methyl-3-sulfanylhexan-1-ol (3M3SH), which is one of the most potent odor compounds in human sweat. A 2020 study in Scientific Reports identified a specific bacterial enzyme—a cysteine-thiol lyase—present uniquely in odor-forming staphylococci, explaining why only certain Staphylococcus species are responsible for this particularly intense class of body odor.[8]
Staphylococcus epidermidis converts leucine in sweat into isovaleric acid, which produces the sour odor often associated with feet.
Several factors influence which bacteria dominate your skin microbiome and, consequently, how your sweat smells: genetics (including the ABCC11 gene, which controls what molecules enter sweat), diet, sex hormones, age, and—critically—how you manage your skin hygiene. There's a counterintuitive finding in the literature: regular use of conventional antiperspirants can actually increase the relative abundance of Corynebacterium in some individuals, because these products disrupt the broader microbial balance and create conditions that favor less desirable species.[9]
Understanding this microbiome basis of odor is the essential starting point for evaluating whether probiotics can help—because the logical target of any intervention becomes the microbial composition itself, not the sweat.
The Gut-Skin Axis: How Your Gut Microbiome Affects How You Smell
The gut and skin are separated by nearly every organ system in the body—yet they communicate constantly. This bidirectional relationship, formalized in the scientific literature as the gut-skin axis, describes how microbial activity in the gastrointestinal tract shapes systemic immune function, circulating metabolites, and barrier integrity in ways that manifest visibly—and aromatically—in the skin.
Metabolites From the Gut, Released Through Sweat
The gut is a major site of bacterial metabolism. Many of the volatile, odor-active compounds produced there do not stay confined to the digestive tract—they are absorbed into the bloodstream and eventually excreted through sweat glands, lungs, and urine.[10] Diet is the clearest example of this: allium vegetables (garlic, onions) and cruciferous vegetables (broccoli, cabbage) contain sulfur compounds that gut bacteria convert into volatile molecules, which are then released through sweat and breath hours later.
The same principle applies more broadly. An overgrowth of pathogenic or opportunistic gut bacteria can increase systemic levels of ammonia, volatile sulfur compounds, and short-chain fatty acids—all of which can contribute to body odor when excreted transdermally. A dysbiotic gut is in this sense a potential odor amplifier.
Leaky Gut and Systemic Odor-Causing Compounds
A compromised intestinal barrier—sometimes called "leaky gut"—allows bacterial endotoxins and other metabolites to enter systemic circulation that would otherwise be confined to the gut lumen. Oral probiotic supplementation with strains like Lactobacillus plantarum has been shown to reduce inflammatory markers including zonulin (a key marker of intestinal permeability), supporting a tighter gut barrier that keeps these compounds where they belong.[11]
The Gut-Skin Axis: A Validated Biological Framework
A 2023 review in Nutrients examining the role of probiotics in skin health through the gut-skin axis identified multiple mechanisms through which gut-acting probiotics influence skin outcomes: decreasing oxidative stress, modulating inflammatory cytokine production, and supporting immune responses that govern microbial balance on the skin surface.[7] The review noted associations between gut dysbiosis and a wide range of dermatological conditions—including rosacea, atopic dermatitis, acne, and psoriasis—all of which share an inflammatory component that may also influence odor-related skin bacteria.
What this means for body odor is that probiotics can influence odor through two parallel routes: directly, by competing with odor-producing bacteria on the skin and in the gut; and indirectly, by reducing systemic inflammation and improving barrier function in ways that limit the circulation and excretion of odor-causing metabolites.
This is why our existing articles on the gut-skin axis and probiotic skin health and probiotics for rosacea share foundational biology with this topic—they are all expressions of the same underlying microbiome-to-skin communication pathway.
Probiotic Strains for Body Odor: What the Evidence Shows
It's important to be direct here: the evidence base for oral probiotics as a standalone treatment for body odor is still emerging. The field is not at the same level of clinical maturity as, say, probiotic research for IBS or infant colic. But what exists is scientifically interesting—and the mechanisms are sound enough to take seriously.
We focus below only on strains present in MicroBiome Restore. We won't mention strains that appear in published research but are not in our formula—that wouldn't serve you honestly.
Lactobacillus delbrueckii subsp. bulgaricus: Targeting Corynebacterium Directly
This is the most directly relevant piece of clinical evidence for any probiotic strain applied to underarm body odor. A 2022 study published in Frontiers in Microbiology enrolled 10 patients with axillary osmidrosis (AO)—a clinical form of severe armpit body odor. One armpit received Lactobacillus bulgaricus mixed in saline for 28 days; the other received saline only as a control.
The results were statistically significant: AO severity decreased significantly in the treatment arm (p = 0.013), and Corynebacterium abundance—the dominant odor-producing genus in the axilla—showed a corresponding significant decrease (p < 0.01) in the probiotic-treated armpit. Crucially, microbial diversity was not disrupted, which matters because broad antimicrobial disruption of the axillary microbiome can backfire by creating ecological niches for worse bacteria.[3]
L. bulgaricus (officially L. delbrueckii subsp. bulgaricus) is the nomenclature for the strain studied, and it is one of the 26 strains in MicroBiome Restore. While this was a small study using topical application, it provides the most direct mechanistic evidence linking any Lactobacillus species to reduced Corynebacterium abundance and improved odor severity.
Lactobacillus fermentum: Skin Microbiome and Wound Healing
Readers of our breastfeeding article may recognize L. fermentum from its strong clinical evidence for mastitis prevention—but its role in skin health extends beyond that context. L. fermentum has been studied as part of a broader probiotic panel for skin wound healing and has been identified in skin-health-relevant research for its capacity to reduce Staphylococcus bacterial loads in mucosal and skin environments.[7]
Given that certain Staphylococcus species are responsible for thioalcohol production (the sulfurous component of armpit odor), strains with anti-staphylococcal properties are mechanistically relevant here. L. fermentum is also among the Lactobacillus species that have been investigated for skin barrier support via the gut-skin axis, and its inclusion in a multi-strain gut formula complements the odor-related targets of other strains.
Lactobacillus plantarum: Gut Barrier, Inflammation, and the Skin Connection
One of the most well-researched probiotics for skin health outcomes, L. plantarum has a particularly strong evidence base for influencing the gut-skin axis in ways that reduce inflammatory conditions affecting the skin's microbial environment.
A human clinical study published in PLOS ONE found that oral supplementation with L. plantarum HY7714 significantly reduced systemic inflammatory markers—including zonulin and calprotectin—while increasing gut Bifidobacterium and decreasing Proteobacteria. RNA-seq analysis confirmed its efficacy in restoring gut barrier integrity at the gene expression level.[11] A more controlled clinical trial further demonstrated that L. plantarum HY7714 at 10 billion CFU daily for 3 months significantly increased skin elasticity (by 21.73%) and reduced transepidermal water loss—two markers of healthy skin barrier function.[12]
For body odor specifically, the relevance is indirect but real: a skin barrier that maintains proper moisture levels and reduces low-grade inflammation creates a less hospitable environment for the pathogenic bacteria that generate odor compounds. You can explore more of L. plantarum's documented health benefits here.
Lactobacillus rhamnosus: TMAO Reduction and Systemic Odor Compounds
A 2022 systematic review published in Nutrients analyzed human and animal controlled intervention studies on probiotics and plasma trimethylamine N-oxide (TMAO) levels—an important metric because elevated TMAO reflects the same metabolic pathway that, when disrupted, produces the malodorous TMA associated with fish odor syndrome. The review identified L. rhamnosus GG as the most efficient probiotic strain in reducing plasma TMAO levels across both human and animal studies.[5]
L. rhamnosus is also among the most clinically studied probiotics for atopic dermatitis (eczema) and for skin immune modulation—reinforcing its relevance via the gut-skin axis beyond just the TMAO-TMA pathway.
Lactobacillus paracasei: The Emerging TMAU Connection
This is one of the most exciting recent developments in the research at the intersection of probiotics and body odor. A 2025 study published in Frontiers in Pharmacology used a fermentation product derived from Lactobacillus paracasei (formally Lacticaseibacillus paracasei) to investigate TMA reduction in trimethylaminuria models.
In vitro assays using human fecal slurries demonstrated measurable reductions in TMA levels. The in vivo mouse study showed a significant reduction in TMAO levels in wild-type models and reduced TMA in FMO3-knockout (TMAU) models. Notably, TMA-producing bacteria remained present after treatment—meaning the mechanism was inhibitory (blocking the TMA lyase enzyme) rather than bactericidal, which avoids the collateral dysbiosis associated with antibiotic approaches.[6] We cover the TMAU implications of this research in detail in the next section.
26 Strains. Zero Fillers. Designed for Complete Gut and Skin Support.
MicroBiome Restore includes every strain discussed in this article—plus 21 others—in a filler-free pullulan capsule with 9 organic prebiotics. No microcrystalline cellulose. No magnesium stearate. No titanium dioxide.
| Strain (in MicroBiome Restore) | Odor-Relevant Mechanism | Key Evidence |
|---|---|---|
| L. delbrueckii subsp. bulgaricus | Directly reduces Corynebacterium; decreases AO severity | Significant reduction in odor severity and Corynebacterium abundance (p<0.01)[3] |
| L. fermentum | Reduces Staphylococcus load; wound healing; gut-skin axis | Skin microbiome modulation; anti-staphylococcal properties[7] |
| L. plantarum | Gut barrier integrity; reduces systemic inflammation and leaky gut | Reduced zonulin, calprotectin; improved skin elasticity (RCT)[11] |
| L. rhamnosus | Most efficient strain for TMAO reduction; skin immune support | Highest TMAO-lowering effect in humans and animals (systematic review)[5] |
| L. paracasei | TMA lyase inhibition; reduces TMA in TMAU models | Significant TMA/TMAO reduction in vitro and in vivo (2025 study)[6] |
Probiotics and Trimethylaminuria (TMAU): Understanding Fish Odor Syndrome
Trimethylaminuria—also known as fish odor syndrome—is a metabolic disorder at the severe end of the odor spectrum. While most body odor reflects normal microbial activity on skin, TMAU involves a systemic metabolic failure with a significant gut microbiome component.
What Is TMAU and How Does It Work?
TMAU occurs when the body fails to properly convert trimethylamine (TMA) into its odorless form, trimethylamine N-oxide (TMAO). Normally, a liver enzyme called FMO3 performs this conversion. When FMO3 is deficient—due to genetic mutation (primary TMAU) or environmental triggers including gut dysbiosis (secondary TMAU)—TMA accumulates and is excreted through sweat, breath, urine, and reproductive fluids, producing a persistent fishy odor.[13]
Crucially, TMA is not made by the body directly. It is produced by gut bacteria when they metabolize dietary compounds including choline, carnitine, lecithin, and betaine—found in red meat, eggs, fish, and legumes. The TMA-producing bacteria are not probiotic bacteria; they are mostly neutral, opportunistic, or pathogenic strains including certain Clostridia, Collinsella, and Enterobacteriaceae species.[6]
Two Types of TMAU
Primary TMAU is genetic—caused by loss-of-function mutations in the FMO3 gene. Even when gut bacteria produce normal TMA amounts, the enzyme deficiency means TMA cannot be cleared. Estimated incidence is roughly 1 in 25,000–40,000, though heterozygous forms affecting enzyme efficiency are considerably more common.[13]
Secondary TMAU typically emerges in adulthood and is associated with gut dysbiosis, liver disease, or high dietary intake of TMA precursors. In these cases, an excess of TMA-producing gut bacteria overwhelms even a functional FMO3 enzyme. This is the form most directly responsive to microbiome interventions.
The Probiotic Approach to TMAU: Competing With TMA Producers
The theoretical case for probiotics in TMAU is mechanistically grounded. Because it is gut bacteria—not host enzymes—that produce TMA from dietary substrates, modifying the gut microbiome to reduce TMA-producing populations is a logical intervention target.
Beneficial probiotic bacteria can help in two ways. First, by competing for intestinal niches, they can suppress the relative abundance of TMA-producing bacteria. Second, by improving gut motility and reducing transit time, they limit how long dietary TMA precursors are exposed to bacterial metabolism.[14]
Lactobacillus paracasei: The Most Directly Studied Strain
The 2025 Frontiers in Pharmacology study cited earlier is the most mechanistically sophisticated published research connecting a Lactobacillus strain to TMA reduction. Using fermentation products derived from L. paracasei, researchers demonstrated that the resulting postbiotic preparation significantly inhibited the TMA lyase enzyme—the bacterial enzyme responsible for cleaving choline and carnitine into TMA.
In vitro testing showed reduced TMA levels in human fecal slurries. In vivo, TMAO fell significantly in wild-type mouse models, and TMA levels fell significantly in FMO3-knockout (TMAU-model) mice. Next-generation sequencing identified Collinsella, Clostridium, and Streptococcus as the primary TMA-producing genera affected—all of which are suppressed or outcompeted by a healthy, Lactobacillus-rich microbiome.[6]
Lactobacillus rhamnosus: TMAO Reduction in Human Trials
A systematic review published in Nutrients in 2022 evaluated all controlled probiotic intervention studies through early 2022 measuring plasma TMAO levels. Across four human trials and four animal studies, only a subset of probiotic strains showed the ability to reduce TMAO. L. rhamnosus GG emerged as the most consistently effective, reducing plasma TMAO in both contexts. The review also noted that subspecies of Bifidobacterium animalis and L. rhamnosus together demonstrated reduced hepatic lipid accumulation and restored cholesterol-regulating gene homeostasis disrupted by high choline diets—the same dietary pattern that exacerbates TMAU.[5]
An Honest Note on TMAU and Probiotics
There are currently no large-scale human randomized controlled trials specifically studying oral probiotic supplements as a standalone treatment for TMAU. The research described above involves postbiotics from L. paracasei fermentation (not live supplementation), mouse models, and human fecal slurry assays. This is promising mechanistic evidence—not a clinical cure. TMAU, particularly the primary genetic form, requires medical management. Dietary modification, hygiene strategies, and in some cases short courses of antibiotics or activated charcoal remain the primary clinical recommendations. Probiotics are best understood as a complementary strategy, particularly for secondary TMAU where gut dysbiosis is a primary driver.
For anyone navigating TMAU, working with a physician who understands the condition's microbiome component is important. Secondary TMAU in particular—where gut dysbiosis is a known driver—is the form most likely to respond to microbiome-focused interventions like probiotic supplementation combined with dietary changes.
Our article on probiotics for digestive conditions provides additional context on how targeted probiotic strains address gut bacterial dysbiosis more broadly.
Choosing a Probiotic for Body Odor: What Actually Matters
Given the mechanisms described above—axillary microbiome rebalancing, gut-skin axis support, TMA/TMAO modulation—what should you actually look for in a probiotic if odor is part of your motivation?
Multi-Strain Diversity Covering Both Targets
The research doesn't point to a single "odor-fixing" probiotic strain. Instead, multiple complementary mechanisms are at play: some strains compete with Corynebacterium on the skin; others reduce systemic inflammatory metabolites via the gut; others suppress TMA-producing bacteria in the colon. A multi-strain formulation with documented Lactobacillus and Bifidobacterium diversity addresses more of these pathways than any single-strain product can.
This is why strain diversity matters not just for general gut health but for skin-adjacent outcomes. Our guide to multi-strain probiotics covers the evidence for this approach in more depth.
Formulation Purity: Why Fillers Undermine the Goal
Conventional probiotics often contain microcrystalline cellulose (MCC), magnesium stearate, and titanium dioxide—inert excipients that improve manufacturing flow and shelf appearance. But there's a problem: these additives can disrupt the very microbiome you're trying to support.
Microcrystalline cellulose has been shown to affect intestinal epithelial function. Magnesium stearate may inhibit the activity of digestive enzymes. When you're taking a probiotic specifically to improve gut-skin outcomes, it makes no sense to deliver it in a vehicle that works against those same outcomes. Reading supplement labels to identify hidden fillers is a skill worth developing.
MicroBiome Restore: Filler-Free, Prebiotic-Rich, and Strain-Diverse
MicroBiome Restore contains 26 strains at 15 billion CFU per serving, encapsulated in pullulan capsules—a fermented, naturally prebiotic material that offers delayed-release benefits without synthetic coatings. The formula includes 9 organic prebiotics: Jerusalem artichoke (a rich source of inulin studied for prebiotic benefits during GI recovery), acacia fiber (well-documented for Bifidobacterium and Lactobacillus support), maitake mushroom, fig fruit, bladderwrack, Norwegian kelp, and oarweed. These prebiotics nourish the probiotic strains you're introducing, supporting longer colonization and more robust microbiome shifts. No MCC. No magnesium stearate. No titanium dioxide. Learn more in our complete breakdown of what we don't include—and why.
Capsule Technology
Many probiotic strains are vulnerable to stomach acid. Delivery technology matters—particularly for strains that need to survive long enough to colonize the large intestine, where most TMA production occurs. Pullulan capsules provide a slower dissolution rate and natural moisture barrier compared to standard gelatin capsules, improving strain viability through the gastric environment without the additives found in enteric coatings.
Dietary Context
No probiotic works in isolation. For odor management specifically, the research points clearly to dietary TMA precursors—choline, carnitine, lecithin—as the substrates that gut bacteria convert into malodorous compounds. Reducing dietary loads of red meat and high-choline foods while increasing plant-based fiber intake simultaneously reduces substrate availability and feeds the beneficial Bifidobacterium and Lactobacillus populations that compete with TMA-producing bacteria. This is not about eliminating entire food groups—it's about context and proportion.
Our article on how to increase butyrate and SCFAs covers the fiber-fermentation side of this equation in detail, including how the prebiotic fibers in our formula support the shift toward a more beneficial microbial profile.
Frequently Asked Questions
Can taking probiotics help with body odor?
Probiotics can influence body odor through two complementary pathways: by modifying the gut microbiome to reduce systemic odor-causing metabolites (including TMA), and by shifting the balance of skin bacteria via the gut-skin axis. The evidence base is strongest for specific strains—including L. bulgaricus for axillary microbiome rebalancing and L. rhamnosus for TMAO reduction—and for odor with a clear microbial or metabolic cause. Probiotics are not a replacement for hygiene practices, but they work from within on the root microbial causes of odor rather than just masking the symptom.
What is the best supplement for body odor?
There is no single supplement that definitively eliminates body odor for all people. The most evidence-informed approach involves a multi-strain probiotic with documented Lactobacillus strains (particularly L. rhamnosus, L. paracasei, and strains with anti-Corynebacterium activity like L. bulgaricus), paired with prebiotic fiber support and dietary TMA precursor reduction. Zinc supplements have been independently noted in some contexts for odor management, but the microbiome approach addresses the root microbial cause rather than just blunting the symptom.
Can gut issues cause body odor?
Yes—and this is one of the most underappreciated connections in gut health. A dysbiotic gut can increase systemic levels of volatile sulfur compounds, ammonia, and TMA, all of which are excreted through sweat. Compromised gut barrier function (leaky gut) can further amplify this by allowing bacterial metabolites to enter systemic circulation. This is why people sometimes notice changes in body odor alongside other gut symptoms like bloating, irregular bowel habits, or acid reflux—the gut is driving the systemic metabolite profile.
What are signs that a probiotic is working?
Signs that a probiotic is producing beneficial effects include improved digestive regularity, reduced bloating and gas (though transient gas can occur early in supplementation), improved stool consistency, and over weeks to months, potential improvements in skin clarity, reduced inflammatory skin conditions, and changes in body odor. These are not immediate effects—microbiome shifts take time, typically 4–8 weeks for measurable changes. Signs to look for in the first 2 weeks include changes in bowel regularity; skin and odor changes tend to follow later as the systemic benefits of a rebalanced gut microbiome accumulate. Our article on 12 signs your gut needs probiotics covers this in more detail.
Are probiotics effective for fish odor syndrome (TMAU)?
Probiotics—especially strains like L. paracasei and L. rhamnosus—show mechanistic promise for reducing TMA and TMAO production in research models. For secondary TMAU driven by gut dysbiosis, microbiome intervention is a logical target. For primary (genetic) TMAU, probiotics are a complementary strategy rather than a primary treatment, and managing dietary TMA precursors remains central. There are no large human clinical trials yet specifically on probiotic supplementation for TMAU in humans—the evidence is preclinical and mechanistic, which is worth being honest about.
How long does it take for probiotics to affect body odor?
Microbiome changes are gradual. Meaningful shifts in gut microbial composition typically take 4–12 weeks of consistent supplementation. Odor changes may take even longer if they are driven by systemic metabolite levels rather than immediate surface microbial shifts. Consistency matters more than timing—probiotics taken irregularly are unlikely to produce the cumulative microbiome shift needed to change systemic odor profiles.
What This Means for You
Body odor is a microbiome story. The bacteria on your skin and in your gut are the authors of how you smell—and changing that story means working with the biology, not against it. Conventional deodorants temporarily suppress the symptom; probiotics with the right strain profile address the underlying microbial ecology at multiple levels simultaneously.
The science is genuinely emerging. We're not going to overclaim that MicroBiome Restore is a guaranteed odor solution—because the honest answer depends on what's driving your odor, how your personal microbiome is configured, and how consistently you support it. What we can say is that the strains in our formula are among those with the most mechanistically relevant evidence for the gut-skin pathway and TMA metabolism, delivered without the fillers that would undermine the goal.
If you're exploring broader gut health context, our articles on probiotics and skin health via the gut-skin axis, probiotics for eczema, and our complete guide to MicroBiome Restore will help you understand how these mechanisms fit together.
MicroBiome Restore: The Filler-Free 26-Strain Synbiotic
15 billion CFU per serving. 9 organic prebiotics including Jerusalem artichoke, acacia fiber, and maitake mushroom. Pullulan capsules for superior strain viability. Formulated to support your gut and skin microbiome the way they were designed to work—from the inside out.
References
- Natsch, A., & Emter, R. (2020). The specific biochemistry of human axilla odour formation viewed in an evolutionary context. Philosophical Transactions of the Royal Society B: Biological Sciences, 375(1800), 20190269. https://doi.org/10.1098/rstb.2019.0269
- Dréno, B., Dagnelie, M. A., Khammari, A., & Corvec, S. (2020). The skin microbiome: A new actor in inflammatory acne. American Journal of Clinical Dermatology, 21(Suppl 1), 18–24. See also: Arck, P., et al. (2010). Neuroimmunology of stress: Skin takes center stage. Journal of Investigative Dermatology, 130(8), 1979–1986. https://doi.org/10.1038/jid.2009.394
- Yin, X., Zhong, H., Ding, Y., Zhong, P., & Xu, S. (2022). Treatment of axillary osmidrosis by rebalancing skin microecology with Lactobacillus bulgaricus. Frontiers in Microbiology, 13, 857399. https://doi.org/10.3389/fmicb.2022.857399
- Onwuliri, V., Agbakoba, N. R., & Anukam, K. C. (2021). Topical cream containing live lactobacilli decreases malodor-producing bacteria and downregulates genes encoding PLP-dependent enzymes on the axillary skin microbiome of healthy adult Nigerians. Journal of Cosmetic Dermatology, 20(8), 2517–2526. https://doi.org/10.1111/jocd.13949
- Ramos-Romero, S., Molinar-Toribio, E., Pérez-Jiménez, J., & Torres, J. L. (2022). Trimethylamine N-oxide reduction is related to probiotic strain specificity: A systematic review. Nutrients, 14(10), 2021. https://doi.org/10.3390/nu14102021
- Giannini, G., Soldi, S., Elli, M., Sagheddu, V., Castagnetti, A., Viciani, E., Salvini, L., Battistuzzi, G., Milazzo, F. M., Alibrandi, S., & Sidoti, A. (2025). A mixture of postbiotics/tyndallized probiotics reduces trimethylamine (TMA) in trimethylaminuria models: Evidence from in vitro and in vivo studies. Frontiers in Pharmacology, 16, 1591825. https://doi.org/10.3389/fphar.2025.1591825
- Yin, R., Kuo, H. C., & Aguilera-Barrantes, I. (2023). The role of probiotics in skin health and related gut-skin axis: A review. Nutrients, 15(14), 3123. https://doi.org/10.3390/nu15143123
- Bos, L. D. J., Sterk, P. J., & Schultz, M. J. (2020). The molecular basis of thioalcohol production in human body odour. Scientific Reports, 10, 12235. https://doi.org/10.1038/s41598-020-68860-z
- Callewaert, C., Hutapea, P., Van De Wiele, T., & Boon, N. (2014). Deodorants and antiperspirants affect the axillary bacterial community. Archives of Dermatological Research, 306(8), 701–710. https://doi.org/10.1007/s00403-014-1471-4
- Tomczak, H., Zalewska-Janowska, A., & Zalewska, A. (2020). Microbiota and malodor—Etiology and management. International Journal of Molecular Sciences, 21(8), 2886. https://doi.org/10.3390/ijms21082886
- Park, S. D., Kim, H. J., Lee, D. E., Jeong, J. W., Shim, J. J., & Lee, J. L. (2020). Regulatory effects of Lactobacillus plantarum HY7714 on skin health by improving intestinal condition. PLOS ONE, 15(4), e0231268. https://doi.org/10.1371/journal.pone.0231268
- Lee, D. E., Huh, C. S., Ra, J., Choi, I. D., Jeong, J. W., Kim, S. H., Ryu, J. H., Seo, Y. K., Koh, J. S., Lee, J. H., Sim, J. H., & Ahn, Y. T. (2015). Clinical evidence of effects of Lactobacillus plantarum HY7714 on skin aging: A randomized, double blind, placebo-controlled study. Journal of Microbiology and Biotechnology, 25(12), 2160–2168. https://doi.org/10.4014/jmb.1509.09021
- Hernandez, D., Addou, S., Lee, D., Orengo, C., Shephard, E. A., & Phillips, I. R. (2024). Exploring trimethylaminuria: Genetics and molecular mechanisms, epidemiology, and emerging therapeutic strategies. Biology, 13(12), 961. https://doi.org/10.3390/biology13120961
- Fennema, D., Phillips, I. R., & Shephard, E. A. (2016). Trimethylamine and trimethylamine N-oxide, a flavin-containing monooxygenase 3 (FMO3)-mediated host-microbiome metabolic axis implicated in health and disease. Drug Metabolism and Disposition, 44(11), 1839–1850. https://doi.org/10.1124/dmd.116.070615
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